CUPRIMINE Capsule 250mg Irlanda - inglês - HPRA (Health Products Regulatory Authority)

cuprimine capsule 250mg

merck sharp and dohme limited - penicillamine - capsule - 250mg

PENICILLAMINE capsule Estados Unidos - inglês - NLM (National Library of Medicine)

penicillamine capsule

ani pharmaceuticals, inc. - penicillamine (unii: gnn1dv99gx) (penicillamine - unii:gnn1dv99gx) - penicillamine capsules are indicated in the treatment of wilson’s disease, cystinuria, and in patients with severe, active rheumatoid arthritis who have failed to respond to an adequate trial of conventional therapy. available evidence suggests that penicillamine capsules are not of value in ankylosing spondylitis. wilson’s disease - wilson’s disease (hepatolenticular degeneration) occurs in individuals who have inherited an autosomal recessive defect that leads to an accumulation of copper far in excess of metabolic requirements. the excess copper is deposited in several organs and tissues, and eventually produces pathological effects primarily in the liver, where damage progresses to postnecrotic cirrhosis, and in the brain, where degeneration is widespread. copper is also deposited as characteristic, asymptomatic, golden-brown kayser-fleischer rings in the corneas of all patients with cerebral symptomatology and some patients who are either asymptomatic or manifest only hepatic symptomatology. two types o

PENICILLAMINE- penicillamine capsule Estados Unidos - inglês - NLM (National Library of Medicine)

penicillamine- penicillamine capsule

actavis pharma, inc. - penicillamine (unii: gnn1dv99gx) (penicillamine - unii:gnn1dv99gx) - penicillamine capsules are indicated in the treatment of wilson's disease, cystinuria, and in patients with severe, active rheumatoid arthritis who have failed to respond to an adequate trial of conventional therapy. available evidence suggests that penicillamine capsules are not of value in ankylosing spondylitis. wilson’s disease (hepatolenticular degeneration) occurs in individuals who have inherited an autosomal recessive defect that leads to an accumulation of copper far in excess of metabolic requirements. the excess copper is deposited in several organs and tissues, and eventually produces pathological effects primarily in the liver, where damage progresses to postnecrotic cirrhosis, and in the brain, where degeneration is widespread. copper is also deposited as characteristic, asymptomatic, golden-brown kayser-fleischer rings in the corneas of all patients with cerebral symptomatology and some patients who are either asymptomatic or manifest only hepatic symptomatology. two types of patients require t