cystopurin - Resultats de la cerca

ani pharmaceuticals, inc. - penicillamine (unii: gnn1dv99gx) (penicillamine - unii:gnn1dv99gx) - penicillamine capsules are indicated in the treatment of wilson’s disease, cystinuria, and in patients with severe, active rheumatoid arthritis who have failed to respond to an adequate trial of conventional therapy. available evidence suggests that penicillamine capsules are not of value in ankylosing spondylitis. wilson’s disease - wilson’s disease (hepatolenticular degeneration) occurs in individuals who have inherited an autosomal recessive defect that leads to an accumulation of copper far in excess of metabolic requirements. the excess copper is deposited in several organs and tissues, and eventually produces pathological effects primarily in the liver, where damage progresses to postnecrotic cirrhosis, and in the brain, where degeneration is widespread. copper is also deposited as characteristic, asymptomatic, golden-brown kayser-fleischer rings in the corneas of all patients with cerebral symptomatology and some patients who are either asymptomatic or manifest only hepatic symptomatology. two types o

Estats Units - anglès - NLM (National Library of Medicine)

penicillamine- penicillamine capsule

actavis pharma, inc. - penicillamine (unii: gnn1dv99gx) (penicillamine - unii:gnn1dv99gx) - penicillamine capsules are indicated in the treatment of wilson's disease, cystinuria, and in patients with severe, active rheumatoid arthritis who have failed to respond to an adequate trial of conventional therapy. available evidence suggests that penicillamine capsules are not of value in ankylosing spondylitis. wilson’s disease (hepatolenticular degeneration) occurs in individuals who have inherited an autosomal recessive defect that leads to an accumulation of copper far in excess of metabolic requirements. the excess copper is deposited in several organs and tissues, and eventually produces pathological effects primarily in the liver, where damage progresses to postnecrotic cirrhosis, and in the brain, where degeneration is widespread. copper is also deposited as characteristic, asymptomatic, golden-brown kayser-fleischer rings in the corneas of all patients with cerebral symptomatology and some patients who are either asymptomatic or manifest only hepatic symptomatology. two types of patients require t

Estats Units - anglès - NLM (National Library of Medicine)

penicillamine tablet, film coated

par pharmaceutical, inc. - penicillamine (unii: gnn1dv99gx) (penicillamine - unii:gnn1dv99gx) - penicillamine tablets are indicated in the treatment of wilson’s disease, cystinuria, and in patients with severe, active rheumatoid arthritis who have failed to respond to an adequate trial of conventional therapy. available evidence suggests that penicillamine tablets are not of value in ankylosing spondylitis. wilson’s disease - wilson’s disease (hepatolenticular degeneration) results from the interaction of an inherited defect and an abnormality in copper metabolism. the metabolic defect, which is the consequence of the autosomal inheritance of one abnormal gene from each parent, manifests itself in a greater positive copper balance than normal. as a result, copper is deposited in several organs and appears eventually to produce pathologic effects most prominently seen in the brain, where degeneration is widespread; in the liver, where fatty infiltration, inflammation, and hepatocellular damage progress to postnecrotic cirrhosis; in the kidney, where tubular and glomerular dysfunction results; and in the

Estats Units - anglès - NLM (National Library of Medicine)

penicillamine capsule

par pharmaceutical, inc. - penicillamine (unii: gnn1dv99gx) (penicillamine - unii:gnn1dv99gx) - penicillamine capsules are indicated in the treatment of wilson's disease, cystinuria, and in patients with severe, active rheumatoid arthritis who have failed to respond to an adequate trial of conventional therapy. available evidence suggests that penicillamine capsules are not of value in ankylosing spondylitis. w il son’s disease — wilson’s disease (hepatolenticular degeneration) occurs in individuals who have inherited an autosomal recessive defect that leads to an accumulation of copper far in excess of metabolic requirements. the excess copper is deposited in several organs and tissues, and eventually produces pathological effects primarily in the liver, where damage progresses to postnecrotic cirrhosis, and in the brain, where degeneration is widespread. copper is also deposited as characteristic, asymptomatic, golden-brown kayser-fleischer rings in the corneas of all patients with cerebral symptomatology and some patients who are either asymptomatic or manifest only hepatic symptomatology.